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Upon ischemic injury in stroke, neuronal HMGB1 is abundantly released into the extracellular space, both by active secretion and passive release, to accumulate in the ischemic core of the acute phase and to activate resident brain immune cells, leading to the recruitment of mononuclear cells that further amplify inflammation (Singh et al., 2016)

Over the last four decades, obesity prevalence in the USA has tripled, now affecting over 42% of the population, and is associated with chronic inflammation and metabolic disturbances [25]

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Q1) and lipid cluster levels LC3, LC4, LC5, LC6, and LC7, their strength of association based on LR models was comparatively weaker (Fig